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Whether BRCA1 and non-BRCA1 breast cancers are associated with different prognoses is controversial and is crucial for the management of unaffected and affected female carriers of germline BRCA1 mutations. Verhoog and colleagues1 have reported a series of familial BRCA1 breastcancer cases, the prognoses of which did not differ from sporadic cases, which is in contrast to previous studies that have suggested a better prognosis for BRCA1-mutationassociated cancers.2 We followed up a series of 123 women with breast cancer at younger than 36 years, diagnosed at the Institut Curie, Paris, between January, 1990, and December, 1995. With their informed consent, they were sampled between January, 1993, and December, 1995, for BRCA1mutation testing.3, 4 We identified 15 likely deleterious BRCA1 mutations with similar nature and gene distribution to those reported in the Breast Cancer Information core database.5 The mean followup was 43 months range 493 ; . Four 27% ; women had no family history of breast or ovarian cancer. Mean age at diagnosis was similar in BRCA1 and non-BRCA1 groups 30 vs 32 years ; . We found no difference in tumour size or axillary-nodal status between the two groups. More BRCA1 than non-BRCA1 carriers had tumours of histological grade 3 71 vs 24%, p 005 ; and more absence of oestrogen and progesterone-receptors 90 vs 37%, p 001; 70 vs 35%, p 005 ; . The number of synchronous and metachronous contralateral tumours was higher in the BRCA1 group 23 vs 4%, p 001 ; . The disease-free survival was non-significantly shorter in the BRCA1 group, contralateral disease was excluded log-rank test on Kaplan-Meier survival probabilities p 019 ; . By contrast, the overall survival was significantly poorer among BRCA1 patients p 004, figure ; , in accordance with a higher frequency of adverse pathobiological features among women with BRCA1 tumours. The significantly poorer prognosis that seemed to be associated with a BRCA1 mutation in our series contrasted with previous studies.1, 2 We saw a difference in overall but not in disease-free survival, which may change with. The CSF, IFN- is detected together with TNF and IL-10 during active MS [54]. IL-2 induces T cell proliferation and differentiation and is mitogenic to oligodendrocytes. Examination of blood, plasma, CSF, and brain tissue from patients with MS demonstrated increased IL-2 levels [10, 55]. IL-12 is detected in active MS lesions and plays a pivotal role in the host's protective immunity against infectious agents, against foreign grafts, and for tumorigenesis. Produced principally by APCs after antigenic challenge and T cell interaction, IL-12 promotes the rapid release of IFN- from T and NK cells and facilitates a biased TH1-type, antigen-specific, cellular immune response. This bias toward TH1-type immunity is determined, at least in part, by the preferential expression of the high-affinity IL-12R by TH1CD4 T cells. IL-12 also drives the preferential switching of antigenspecific immunoglobulins toward IgG2a and IgG2b isotypes, which may enhance phagocytic opsonization, complement fixation, and ADCC [55]. In this way, IL-12 may play a protective role in disease. Indeed, IL-12 produced from keratinocytes exposed to ultraviolet B light might actually be a protective mechanism against MS.

The General Meeting, voting on the quorum and majority conditions for Ordinary Meetings, having reviewed the Statutory Auditors' Special Report on agreements covered by articles L.225-38 et seq of the Commercial Code entered into and performed during the year, approves all parts of this report and the agreements described therein and septra.
Defined. This case report concerns an unusual case of Castleman's disease mimicking a pancreatic tumour with a rapidly fatal outcome. Case report A 56-year-old hypertensive woman was admitted to our hospital because of fatigue, weight loss and vague abdominal pain of one-month duration. The patient had no significant prior illnesses. On admission, physical examination and laboratory findings, including tumour markers and inflammatory mediators, revealed nothing in particular. Abdominal ultrasound demonstrated a well-defined isolated, 3, 7 1, cm, hypoechoic mass in the pancreatic head. The abdominal computed tomographic scan confirmed the presence of the mass by revealing the anterior lobulation of the pancreatic head. This appearance mimicked the mass lesion, which was isodense with the pancreatic paranchyma. The pancreatic duct was not dilated. No enlarged abdominal or retroperitoneal lymph nodes were detected Fig. 1 ; . The initial diagnosis was pancreatic cancer and the case underwent surgical excision. Exploratory laparotomy revealed a smooth, solid mass, 3 cm in diameter, located in the pancreatic body. A Whipple procedure was performed. Gross examination showed a tumour 2, 5 2, cm size without adhesion to the surrounding tissue. Histopathologically, the tissue specimen revealed an angiofollicular lymph node hyperplasia Castleman's disease ; of the hyaline vascular type Figs. 2 and 3 ; . The patient had an uneventful early postoperative course and was discharged 14 days postoperatively. Her symptoms relieved and healed postoperatively, but unfortunately she did not attend her follow-up appointments after the third month.

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Customer contact: Adriana Martinelli, Education Projects Coordinator, Ayrton Senna Foundation Mavi Simo, Coordinator, Canal Futura, Roberto Marinho Foundation Please contact the Partners in Learning Manager first ; "By mixing the content developed for the programs with real cases already happening in schools, we can clearly demonstrate the concepts and best practices of information and communication technology in schools." Adriana Martinelli, Education Projects Coordinator, Ayrton Senna Foundation and skelaxin. 16 Tamargo Surgeons Annual Meeting, Minneapolis, Minnesota, April 1996. Journal of Neurosurgery. 84: 323, 1996. Walter, K. A., T. F. Witham, and R. J. Tamargo. The Glasgow Coma Scale: A better system for grading aneurysmal subarachnoid hemorrhage? American Association of Neurological Surgeons Annual Meeting, Minneapolis, Minnesota, April 1996. Journal of Neurosurgery. 84: 320, 1996. O'Malley, B. W., Jr., and R. J. Tamargo. Anterior cranial base reconstruction using a split calvarial bone graft. Second International Skull Base Congress, San Diego, California, June 1996. 39. Takagi, M., D. S. Zee, and R. J. Tamargo. Effect of dorsal cerebellar vermal lesions on saccades and pursuit in monkeys. Society for Neuroscience, Washington, D. C., November 1996. Society for Neuroscience Abstracts. 22 2 ; : 1458, 1996. 40. Oshiro, E. M., and R. J. Tamargo. Sustainedrelease of urokinase from a polymer preparation: an in vitro evaluation of release kinetics and biological activity. 22nd International Joint Conference on Stroke and Cerebral Circulation, American Heart Association, Anaheim, California, February 1997. Stroke 28: 263, 1997. Thai, Q.A., E. M. Oshiro, and R. J. Tamargo. Inhibition of posthemorrhagic vasospasm using ibuprofenloaded polymers. 22nd International Joint Conference on Stroke and Cerebral Circulation, American Heart Association, Anaheim, California, February 1997. Stroke 28: 263, 1997. Oshiro, E. M., D. A. Rini, and R. J. Tamargo. Contralateral approaches to bilateral cerebral aneurysms: A surgical case series and microsurgical cadaver study. Annual Meeting of the Joint Section on Cerebrovascular Surgery, Anaheim, California, February 1997. 43. Javedan, S. P., and R. J. Tamargo. The diagnostic yield of brain biopsy in neurodegenerative disorders. American Association of Neurological Surgeons Annual Meeting, Denver, Colorado, April 1997. Journal of Neurosurgery. 86: 362A, 1997. Thai, Q.A., E. M. Oshiro, and R. J. Tamargo. Inhibition of posthemorrhagic vasospasm using ibuprofenloaded controlledrelease polymers. American Association of Neurological Surgeons Annual Meeting, Denver, Colorado, April 1997. Journal of Neurosurgery. 86: 336, 1997. Oshiro, E. M., and R. J. Tamargo. Sustainedrelease of urokinase from a polymer preparation: An in vitro evaluation of release kinetics and biological activity. American Association of Neurological Surgeons Annual Meeting, Denver, Colorado, April 1997. Journal of Neurosurgery. 86: 335, 1997.

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